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156-915-1 - Accelerated CCSE 1.1 NGX - BrainDump Information

Vendor Name : CheckPoint
Exam Code : 156-915-1
Exam Name : Accelerated CCSE 1.1 NGX
Questions and Answers : 272 Q & A
Updated On : November 15, 2018
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156-915-1 exam Dumps Source : Accelerated CCSE 1.1 NGX

Test Code : 156-915-1
Test Name : Accelerated CCSE 1.1 NGX
Vendor Name : CheckPoint
Q&A : 272 Real Questions

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Incyte and Merck supply extra particulars on up to now introduced Collaboration Investigating Epacadostat and KEYTRUDA ® (pembrolizumab) | killexams.com Real Questions and Pass4sure dumps

WILMINGTON, Del. & KENILWORTH, N.J.--(enterprise WIRE)--Incyte agency (Nasdaq:INCY) and Merck (NYSE:MRK), called MSD outdoor the USA and Canada, nowadays announced extra details of their medical development program investigating the aggregate of epacadostat, Incyte’s investigational oral selective IDO1 enzyme inhibitor, with KEYTRUDA® (pembrolizumab), Merck’s anti-PD-1 remedy, in sufferers throughout five tumor types: metastatic melanoma, non-small cellphone lung melanoma (NSCLC), bladder melanoma, renal cell carcinoma (RCC), and squamous phone carcinoma of the top and neck (SCCHN).

The extended collaboration now includes seven pivotal studies:

  • A part three study in metastatic melanoma (trial underway);
  • two section 3 reviews in first-line NSCLC, including each PD-L1 excessive-expressing and PD-L1 unselected populations;
  • two section 3 experiences in bladder melanoma, together with one in first-line bladder cancer and one in 2d-line bladder melanoma;
  • a part 3 look at in first-line RCC; and
  • a section 3 examine in first-line SCCHN.
  • Incyte and Merck will share obligations for funding these pivotal studies, and Merck should be chargeable for conducting the rigors.

    “we are joyful to announce further details of our collaboration with Merck and to be moving forward with diverse phase 3 reviews in these 4 additional tumor kinds as at once as feasible,” noted Steven Stein, M.D., Chief clinical Officer, Incyte. “We look ahead to our endured medical collaboration, the initiation of those further pivotal trials and to further exploring the expertise of this novel immune-remedy combination as a medicine for sufferers with cancer.”

    “Our clinical trial software with Incyte represents a vital part of our multi-pronged efforts to investigate the expertise for KEYTRUDA (pembrolizumab) in combination with promising compounds, equivalent to epacadostat,” referred to Dr. Roy Baynes, senior vice president, head of medical construction, and Chief clinical Officer, Merck analysis Laboratories. “via these trials, our long-term intention is to aid make a change within the lives of people with a wide array of cancers.”

    About ECHO

    The ECHO medical trial application was dependent to investigate the efficacy and protection of epacadostat as a core part of combination remedy in oncology. Ongoing section 1 and part 2 reviews evaluating epacadostat in mixture with PD-1 and PD-L1 inhibitors together plan to join over 900 patients in a huge latitude of strong tumor forms as well as hematological malignancies. ECHO-301 (NCT02752074), a phase 3 randomized, double-blind, placebo-managed look at investigating KEYTRUDA in aggregate with epacadostat or placebo for the primary-line remedy of sufferers with superior or metastatic melanoma, is also underway.

    About Epacadostat (INCB024360)

    Indoleamine 2,3-dioxygenase 1 (IDO1) is a key immunosuppressive enzyme that modulates the anti-tumor immune response via advertising regulatory T mobile generation and blocking effector T mobilephone activation, thereby facilitating tumor increase by means of allowing melanoma cells to avoid immune surveillance. Epacadostat is a primary-in-category, tremendously potent and selective oral inhibitor of the IDO1 enzyme that reverses tumor-associated immune suppression and restores valuable anti-tumor immune responses. In single-arm experiences, the combination of epacadostat and immune checkpoint inhibitors has proven proof-of-idea in sufferers with unresectable or metastatic melanoma. In these reviews, epacadostat mixed with the CTLA-4 inhibitor ipilimumab or the PD-1 inhibitor KEYTRUDA superior response prices compared with stories of the immune checkpoint inhibitors on my own.

    About KEYTRUDA®(pembrolizumab)

    KEYTRUDA is a humanized monoclonal antibody that works by way of expanding the capability of the body’s immune gadget to help observe and battle tumor cells. KEYTRUDA blocks the interplay between PD-1 and its ligands, PD-L1 and PD-L2, thereby activating T lymphocytes which may have an effect on both tumor cells and in shape cells.

    KEYTRUDA is run as an intravenous infusion over 30 minutes each three weeks for the accepted signals. KEYTRUDA for injection is provided in a one hundred mg single-dose vial.

    KEYTRUDA (pembrolizumab) signs and Dosing

    Melanoma

    KEYTRUDA is indicated for the medication of patients with unresectable or metastatic melanoma at a dose of 2 mg/kg every three weeks unless sickness development or unacceptable toxicity.

    Lung melanoma

    KEYTRUDA (pembrolizumab) is indicated for the first-line treatment of sufferers with metastatic non-small mobile lung cancer (NSCLC) whose tumors have excessive PD-L1 expression [tumor proportion score (TPS) ≥50%] as decided via an FDA-authorised test, with out a EGFR or ALK genomic tumor aberrations.

    KEYTRUDA is additionally indicated for the medication of sufferers with metastatic NSCLC whose tumors specific PD-L1 (TPS ≥1%) as determined by means of an FDA-approved examine, with disorder development on or after platinum-containing chemotherapy. sufferers with EGFR or ALK genomic tumor aberrations should have disorder progression on FDA-permitted therapy for these aberrations in advance of receiving KEYTRUDA.

    In metastatic NSCLC, KEYTRUDA is run at a set dose of 200 mg each three weeks until sickness progression, unacceptable toxicity, or up to 24 months in sufferers with out disease progression.

    Head and Neck melanoma

    KEYTRUDA is indicated for the remedy of patients with recurrent or metastatic head and neck squamous cell carcinoma (HNSCC) with ailment progression on or after platinum-containing chemotherapy. This indication is accredited under accelerated approval in line with tumor response fee and sturdiness of response. persevered popularity of this indication may well be contingent upon verification and description of clinical benefit within the confirmatory trials. In HNSCC, KEYTRUDA is administered at a set dose of 200 mg each three weeks until ailment progression, unacceptable toxicity, or up to 24 months in patients without disease development.

    Classical Hodgkin Lymphoma

    KEYTRUDA is indicated for the remedy of grownup and pediatric sufferers with refractory classical Hodgkin lymphoma (cHL), or who have relapsed after three or greater prior strains of therapy. This indication is authorized beneath accelerated approval in accordance with tumor response fee and sturdiness of response. persisted acclaim for this indication may be contingent upon verification and outline of medical benefit in the confirmatory trials. In adults with cHL, KEYTRUDA is run at a hard and fast dose of 200 mg every three weeks until disease development or unacceptable toxicity, or as much as 24 months in patients devoid of disorder progression. In pediatric sufferers with cHL, KEYTRUDA is run at a dose of two mg/kg (as much as a optimum of 200 mg) every three weeks until sickness development or unacceptable toxicity, or as much as 24 months in sufferers without disease development.

    chosen crucial safety assistance for KEYTRUDA® (pembrolizumab)

    KEYTRUDA may cause immune-mediated pneumonitis, including deadly situations. Pneumonitis took place in ninety four (three.4%) of 2799 patients receiving KEYTRUDA, including Grade 1 (0.eight%), 2 (1.3%), 3 (0.9%), four (0.three%), and 5 (0.1%) pneumonitis, and passed off greater generally in sufferers with a background of prior thoracic radiation (6.9%) in comparison to those devoid of (2.9%). computer screen sufferers for signs and signs of pneumonitis. evaluate suspected pneumonitis with radiographic imaging. Administer corticosteroids for Grade 2 or stronger pneumonitis. Withhold KEYTRUDA (pembrolizumab) for Grade 2; completely discontinue KEYTRUDA for Grade three or 4 or recurrent Grade 2 pneumonitis.

    KEYTRUDA may cause immune-mediated colitis. Colitis passed off in 48 (1.7%) of 2799 patients receiving KEYTRUDA, together with Grade 2 (0.four%), 3 (1.1%), and 4 (<0.1%) colitis. computer screen sufferers for signs and signs of colitis. Administer corticosteroids for Grade 2 or more desirable colitis. Withhold KEYTRUDA for Grade 2 or three; permanently discontinue KEYTRUDA for Grade 4 colitis.

    KEYTRUDA may cause immune-mediated hepatitis. Hepatitis came about in 19 (0.7%) of 2799 patients receiving KEYTRUDA, together with Grade 2 (0.1%), 3 (0.four%), and four (<0.1%) hepatitis. display screen patients for adjustments in liver function. Administer corticosteroids for Grade 2 or more desirable hepatitis and, in accordance with severity of liver enzyme elevations, withhold or discontinue KEYTRUDA.

    KEYTRUDA can cause hypophysitis. Hypophysitis happened in 17 (0.6%) of 2799 patients receiving KEYTRUDA, including Grade 2 (0.2%), 3 (0.three%), and four (<0.1%) hypophysitis. monitor patients for indications and symptoms of hypophysitis (including hypopituitarism and adrenal insufficiency). Administer corticosteroids and hormone replacement as clinically indicated. Withhold KEYTRUDA for Grade 2; withhold or discontinue for Grade three or four hypophysitis.

    KEYTRUDA may cause thyroid issues, together with hyperthyroidism, hypothyroidism, and thyroiditis. Hyperthyroidism befell in ninety six (3.4%) of 2799 sufferers receiving KEYTRUDA, including Grade 2 (0.eight%) and 3 (0.1%) hyperthyroidism. Hypothyroidism came about in 237 (eight.5%) of 2799 patients receiving KEYTRUDA, together with Grade 2 (6.2%) and three (0.1%) hypothyroidism. The incidence of new or worsening hypothyroidism become higher in sufferers with HNSCC, occurring in 28 (15%) of 192 patients with HNSCC, including Grade 3 (0.5%) hypothyroidism. Thyroiditis befell in sixteen (0.6%) of 2799 sufferers receiving KEYTRUDA, together with Grade 2 (0.3%) thyroiditis. video display sufferers for adjustments in thyroid characteristic (firstly of medication, periodically all through medicine, and as indicated in line with clinical contrast) and for scientific signals and symptoms of thyroid problems. Administer substitute hormones for hypothyroidism and control hyperthyroidism with thionamides and beta-blockers as acceptable. Withhold or discontinue KEYTRUDA for Grade three or 4 hyperthyroidism.

    KEYTRUDA may cause type 1 diabetes mellitus, including diabetic ketoacidosis, which have been pronounced in 6 (0.2%) of 2799 patients. video display patients for hyperglycemia or other indications and symptoms of diabetes. Administer insulin for classification 1 diabetes, and withhold KEYTRUDA and administer antihyperglycemics in patients with severe hyperglycemia.

    KEYTRUDA can cause immune-mediated nephritis. Nephritis occurred in 9 (0.3%) of 2799 patients receiving KEYTRUDA, including Grade 2 (0.1%), three (0.1%), and 4 (<0.1%) nephritis. computer screen patients for changes in renal characteristic. Administer corticosteroids for Grade 2 or greater nephritis. Withhold KEYTRUDA for Grade 2; permanently discontinue KEYTRUDA for Grade three or four nephritis.

    KEYTRUDA can cause other clinically critical immune-mediated antagonistic reactions. For suspected immune-mediated adversarial reactions, make certain sufficient evaluation to confirm etiology or exclude other motives. in accordance with the severity of the hostile response, withhold KEYTRUDA and administer corticosteroids. Upon development to Grade 1 or much less, provoke corticosteroid taper and continue to taper over at the least 1 month. in keeping with restricted statistics from medical studies in patients whose immune-linked adversarial reactions could not be controlled with corticosteroid use, administration of other systemic immunosuppressants may also be regarded. Resume KEYTRUDA (pembrolizumab) when the adversarial reaction remains at Grade 1 or less following corticosteroid taper. completely discontinue KEYTRUDA for any Grade three immune-mediated antagonistic response that recurs and for any existence-threatening immune-mediated opposed reaction.

    here clinically enormous immune-mediated adverse reactions happened in less than 1% (until otherwise indicated) of 2799 sufferers: arthritis (1.5%), exfoliative dermatitis, bullous pemphigoid, rash (1.four%), uveitis, myositis, Guillain-Barré syndrome, myasthenia gravis, vasculitis, pancreatitis, hemolytic anemia, and partial seizures arising in a affected person with inflammatory foci in brain parenchyma. in addition, myelitis and myocarditis have been reported in other scientific trials, together with cHL, and postmarketing use.

    solid organ transplant rejection has been pronounced in postmarketing use of KEYTRUDA. treatment with KEYTRUDA may additionally boost the possibility of rejection in solid organ transplant recipients. consider the benefit of remedy with KEYTRUDA vs the chance of feasible organ rejection in these patients.

    KEYTRUDA can cause extreme or lifestyles-threatening infusion-linked reactions, which had been pronounced in 6 (0.2%) of 2799 patients. computer screen sufferers for indications and symptoms of infusion-related reactions, including rigors, chills, wheezing, pruritus, flushing, rash, hypotension, hypoxemia, and fever. For Grade 3 or four reactions, cease infusion and completely discontinue KEYTRUDA.

    Immune-mediated issues, including fatal routine, happened in patients who underwent allogeneic hematopoietic stem cell transplantation (HSCT) after being handled with KEYTRUDA. Of 23 patients with cHL who proceeded to allogeneic HSCT after medication with KEYTRUDA on any trial, 6 sufferers (26%) developed graft-versus-host-ailment (GVHD), one among which was fatal, and a couple of sufferers (9%) developed severe hepatic veno-occlusive ailment (VOD) after reduced-depth conditioning, one in every of which became fatal. instances of fatal hyperacute GVHD after allogeneic HSCT have also been reported in patients with lymphoma who acquired a PD-1 receptor-blockading antibody before transplantation. These issues may additionally happen regardless of intervening therapy between PD-1 blockade and allogeneic HSCT. observe sufferers intently for early proof of transplant-connected problems reminiscent of hyperacute GVHD, extreme (Grade three to 4) acute GVHD, steroid-requiring febrile syndrome, hepatic VOD, and different immune-mediated adversarial reactions, and intervene right now.

    in keeping with its mechanism of action, KEYTRUDA can cause fetal harm when administered to a pregnant woman. If used during pregnancy, or if the patient becomes pregnant all over remedy, apprise the affected person of the knowledge hazard to a fetus. suggest adult females of reproductive capabilities to use highly effective contraception all through remedy and for four months after the final dose of KEYTRUDA.

    In KEYNOTE-006, KEYTRUDA changed into discontinued because of adversarial reactions in 9% of 555 sufferers with advanced melanoma; adverse reactions leading to discontinuation in more than one affected person had been colitis (1.4%), autoimmune hepatitis (0.7%), allergic reaction (0.4%), polyneuropathy (0.four%), and cardiac failure (0.four%). opposed reactions resulting in interruption of KEYTRUDA occurred in 21% of patients; the most average (≥1%) became diarrhea (2.5%). the most normal hostile reactions with KEYTRUDA vs ipilimumab were fatigue (28% vs 28%), diarrhea (26% with KEYTRUDA), rash (24% vs 23%), and nausea (21% with KEYTRUDA). Corresponding incidence quotes are listed for ipilimumab best for these adverse reactions that took place at the identical or lower cost than with KEYTRUDA (pembrolizumab).

    KEYTRUDA become discontinued due to adversarial reactions in 8% of 682 patients with metastatic NSCLC. essentially the most common adversarial event resulting in permanent discontinuation of KEYTRUDA became pneumonitis (1.8%). hostile reactions resulting in interruption of KEYTRUDA occurred in 23% of sufferers; probably the most typical (≥1%) have been diarrhea (1%), fatigue (1.three%), pneumonia (1%), liver enzyme elevation (1.2%), reduced urge for food (1.3%), and pneumonitis (1%). essentially the most standard adversarial reactions (taking place in as a minimum 20% of patients and at a much better incidence than with docetaxel) had been decreased appetite (25% vs 23%), dyspnea (23% vs 20%), and nausea (20% vs 18%).

    KEYTRUDA turned into discontinued because of adversarial reactions in 17% of 192 patients with HNSCC. critical antagonistic reactions befell in 45% of sufferers. probably the most frequent critical opposed reactions said in at least 2% of patients had been pneumonia, dyspnea, confusional state, vomiting, pleural effusion, and respiratory failure. the most normal adverse reactions (suggested in at least 20% of sufferers) were fatigue, lowered appetite, and dyspnea. adverse reactions happening in patients with HNSCC have been frequently akin to these taking place in patients with melanoma or NSCLC, aside from accelerated incidences of facial edema (10% all Grades; 2.1% Grades three or four) and new or worsening hypothyroidism.

    KEYTRUDA became discontinued due to adversarial reactions in 5% of 210 patients with cHL and medicine become interrupted as a result of hostile reactions in 26% of patients. Fifteen % (15%) of sufferers had an opposed response requiring systemic corticosteroid remedy. severe antagonistic reactions passed off in 16% of patients. the most ordinary severe adverse reactions (≥1%) covered pneumonia, pneumonitis, pyrexia, dyspnea, GVHD, and herpes zoster. Two sufferers died from reasons apart from disease development; one from GVHD after subsequent allogeneic HSCT and one from septic shock. the most common adversarial reactions (happening in ≥20% of patients) were fatigue (26%), pyrexia (24%), cough (24%), musculoskeletal pain (21%), diarrhea (20%), and rash (20%).

    It isn't well-known no matter if KEYTRUDA is excreted in human milk. as a result of many medicine are excreted in human milk, coach women to discontinue nursing all over treatment with KEYTRUDA and for four months after the last dose.

    There is restricted adventure in pediatric patients. In a examine of forty pediatric sufferers with superior melanoma, PD-L1–fine advanced, relapsed, or refractory solid tumors or lymphoma, patients were handled with KEYTRUDA for a median of 43 days (range 1 to 414 days), with 24 patients (60%) receiving treatment for 42 days or more. The security profile in pediatric patients changed into akin to that viewed in adults handled with KEYTRUDA. Toxicities that happened at a more robust rate (≥15% difference) in these patients when in comparison to adults beneath 65 years of age had been fatigue (forty five%), vomiting (38%), belly ache (28%), hypertransaminasemia (28%), and hyponatremia (18%).

    About Incyte

    Incyte business enterprise is a Wilmington, Delaware-based biopharmaceutical company concentrated on the discovery, development and commercialization of proprietary therapeutics. For additional info on Incyte, please talk over with the company’s site at www.incyte.com.

    comply with @Incyte on Twitter at https://twitter.com/Incyte.

    Merck’s focus on melanoma

    Our intention is to translate breakthrough science into imaginitive oncology drug treatments to help people with cancer global. At Merck, helping americans fight cancer is our ardour and assisting accessibility to our melanoma medicines is our dedication. Our focus is on pursuing research in immuno-oncology and we are accelerating each step within the adventure – from lab to health center – to probably carry new hope to individuals with cancer.

    As part of our focal point on melanoma, Merck is committed to exploring the abilities of immuno-oncology with some of the fastest-growing to be construction courses within the industry. we are presently executing an expansive analysis application that comprises greater than 450 medical trials evaluating our anti-PD-1 therapy across greater than 30 tumor forms. We also proceed to beef up our immuno-oncology portfolio via strategic acquisitions and are prioritizing the construction of a few promising immunotherapeutic candidates with the abilities to enhance the medicine of advanced cancers.

    For more guidance about our oncology medical trials, consult with www.merck.com/clinicaltrials.

    About Merck

    For over a century, Merck has been a worldwide fitness care chief working to aid the area be well. Merck is referred to as MSD backyard the us and Canada. through our prescription drugs, vaccines, biologic cures, and animal fitness items, we work with consumers and operate in additional than 140 countries to bring inventive health options. We also reveal our dedication to increasing access to health care through far-achieving policies, classes and partnerships. For extra tips, talk over with www.merck.com and connect with us on Twitter, fb, YouTube and LinkedIn.

    forward-looking commentary of Incyte enterprise

    except for the ancient information set forth herein, the concerns set forth during this press unlock contain predictions, estimates and other forward-looking statements, including with out difficulty statements concerning: no matter if and when the deliberate pivotal trials investigating epacadostat with KEYTRUDA in any of non-small mobile lung, renal, bladder or head and neck cancers will begin; no matter if any of those experiences will cause any products that could be approved to be used in people anywhere; no matter if these planned collaborations will support enhance clinical consequences for sufferers; even if and when any statistics from the ECHO software could be available.

    These forward-searching statements are subject to dangers and uncertainties that may cause genuine consequences to differ materially, including unanticipated tendencies in and risks involving: the efficacy or safety of the Incyte’s building pipeline; the effects of extra analysis and construction; the high diploma of chance and uncertainty linked to drug construction, scientific trials and regulatory approval processes, other market or economic components and competitive and technological advances; the possibility that results of medical trials may be unsuccessful or inadequate to fulfill relevant regulatory requisites or warrant continued building; our potential to compete in opposition t events with stronger monetary or different elements; better than expected expenses; and such other hazards specific sometimes in Incyte’s studies filed with the Securities and exchange commission, together with our form 10-ok for the yr ending December 31, 2016. Incyte disclaims any intent or responsibility to update these ahead-looking statements.

    ahead-looking statement of Merck & Co., Inc., Kenilworth, N.J., united states of america

    This news unlock of Merck & Co., Inc., Kenilworth, N.J., usa (the “enterprise”) contains “forward-looking statements” inside the meaning of the secure harbor provisions of the U.S. private Securities Litigation Reform Act of 1995. These statements are based upon the present beliefs and expectations of the business’s management and are area to giant dangers and uncertainties. There can be no guarantees with recognize to pipeline items that the items will receive the vital regulatory approvals or that they'll show to be commercially a success. If underlying assumptions prove inaccurate or dangers or uncertainties materialize, precise results may fluctuate materially from these set forth in the ahead-searching statements.

    hazards and uncertainties include however aren't confined to, regular industry conditions and competitors; universal economic elements, including pastime fee and forex change price fluctuations; the have an effect on of pharmaceutical industry rules and fitness care law within the united states and internationally; global trends towards fitness care can charge containment; technological advances, new products and patents attained by way of opponents; challenges inherent in new product construction, including acquiring regulatory approval; the business’s capacity to precisely predict future market situations; manufacturing difficulties or delays; monetary instability of international economies and sovereign risk; dependence on the effectiveness of the company’s patents and different protections for innovative products; and the exposure to litigation, including patent litigation, and/or regulatory moves.

    The enterprise undertakes no responsibility to publicly update any forward-looking observation, even if because of new counsel, future activities or otherwise. further elements that might cause consequences to differ materially from those described in the ahead-searching statements may also be found in the business’s 2016 Annual report on kind 10-okay and the enterprise’s other filings with the Securities and change commission (SEC) obtainable at the SEC’s cyber web web site (www.sec.gov).

    Please see Prescribing assistance for KEYTRUDA (pembrolizumab) at http://www.merck.com/product/usa/pi_circulars/okay/keytruda/keytruda_pi.pdf and affected person advice/medication e book for KEYTRUDA at http://www.merck.com/product/country/pi_circulars/okay/keytruda/keytruda_mg.pdf.




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    With New Turing, NVIDIA Doubles Down on the Future of Real-Time Ray-Tracing | killexams.com real questions and Pass4sure dumps

    18 Oct, 2018 By: Alex Herrera And CAD professionals are expected to reap the benefits.

    NVIDIA's long-expected successor to its Pascal GPU architecture for gaming and professional graphics is here. In August, the GPU developer pulled the covers off Turing, which one could argue is both a successor to not one but both of its preceding generations of graphics processing units — 2016's Pascal and 2017's Volta. In the process, the company confirmed several of the more expected 3D graphics advancements for its next flagship GPU. But it also revealed a few surprises, representing an aggressive-but-justified departure from past generations' decisions about how it formulates products that today are destined for a far wider spectrum of applications than those of years past.

    Perhaps even more significant is the inflection point in GPU evolution that Turing marks, a unification — if not permanent and all-encompassing, at the very least meaningful — of the previous disparate and often conflicting priorities between the GPU's traditional 3D graphics markets and the hot emerging opportunities attracting the company's attention. With Turing, NVIDIA confirms two realizations very much reflected in the GPU's DNA: one, that machine learning is now a valid and justified tool to enhance 3D visual computing; and two, that the time is ripe to begin the long-awaited transition from 3D raster graphics to the ultimate in rendering, real-time ray-tracing.

    Turing, RTX, and NGX

    NVIDIA is finding more ways to leverage machine learning to improve performance and quality for traditional 3D graphics. It's been a while since NVIDIA shaped new GPU architectures and technology strictly for the benefit of traditional raster-based 3D graphics that CAD applications and users have primarily relied upon. Over the past decade, NVIDIA GPUs have pushed well beyond that core space and into high-performance computation ("compute"), autonomous vehicles, robotics, supercomputing, and now, front-and-center, machine learning. And each new generation has walked a careful balance, supporting new applications without handicapping the GPU for its bread-and-butter 3D graphics markets.

    With Turing, NVIDIA made many of the more conventional improvements to its fundamental 3D graphics programmable shader engine, the Streaming Multiprocessor (SM), especially in terms of critical resources like chip registers and cache, and dialed up supporting infrastructure including external memory bandwidth — all good things that contribute to faster, higher-quality interactive 3D graphics crucial to improving the CAD experience and productivity. But those tweaks represent the more expected steps along the tried-and-true GPU evolution path, taking on cost and complexity for features and performance the company is pretty darn sure ISVs and end users alike will value in the near term, if not immediately. More noteworthy than the more conventional 3D graphics features Turing added is what it didn't subtract from the company's previous compute/artificial intelligence (AI)–focused GPU, Volta. With Turing, NVIDIA architects not only didn't strip out Volta's Tensor Cores, they improved on them — and doubled down on the pursuit of real-time rendering by boosting ray-trace-specific acceleration.

    Tensor Cores accelerated AI for ray-tracing — and now they speed conventional 3D raster graphics to boot. Volta's most noteworthy advancement was the inclusion of Tensor Cores, new hardware engines — of significant incremental chip cost (i.e. transistors/silicon area) — to accelerate processing of deep neural networks (DNNs), the lifeblood of machine learning applications. Now, given Volta's primary focus on high-performance computing rather than 3D graphics, the choice to take on the silicon cost of Tensor Cores was certainly novel, but not particularly contentious.

    But unlike Volta and its compute focus, Turing is a graphics-first architecture, so NVIDIA's decision to keep Tensor Cores in Turing raises a very pertinent question: Why would the company dedicate significant cost in a graphics-focused GPU to a feature that doesn't directly benefit graphics? Well, the answer is most interesting and fortunate: That old premise is no longer true, and NVIDIA is now finding compelling ways to leverage machine learning to improve the quality and performance of 3D imagery.

    As discussed in detail in a previous column, "What Does NVIDIA's Ray Tracing News Mean for the CAD Market?" NVIDIA figured out a way to leverage deep learning to significantly improve the performance of ray-traced 3D rendering. Specifically, RTX software exploits Tensor Core hardware incorporated in a DNN in the ray-tracer to accelerate image "convergence" by decreasing the computational load in the latter stages of rendering. Once the image converges into something it can recognize, AI fills in remaining rays/pixels, intelligently de-noising the image and wrapping up the time-consuming rendering process far faster than is possible via exhaustive, full-resolution ray processing.

    RTX technology on Volta (right side) accelerates ray-tracing through machine learning. (Source: NVIDIA)RTX technology (right side) accelerates ray-tracing through AI-accelerated de-noising, compared with the same number of rays without de-noising (left).  (Source: NVIDIA)

    No doubt enthused by the successful synergy of machine learning and graphics with ray-trace processing, NVIDIA researchers began exploring other ways to extract more visual processing goodness out of its GPUs' AI prowess. Extending on the use of DNNs for ray-trace de-noising, NVIDIA unveiled NGX technology, comprised of an expanded set of DNN-driven image-enhancement features.

    The most relevant and compelling example using Turing/NGX to enhance conventional 3D graphics is Deep Learning Super Sampling (DLSS). Essentially, DLSS benefits from a Tensor Core–accelerated DNN that substitutes the usual brute-force pixel super-sampling with intelligent choices based on the scene geometry, and based both on one frame instance and interframe temporal changes. The benefit is that quality improves at the same performance level, or likely more interesting for most applications (because resolution-dependent quality is pretty darn good at this point), performance increases significantly at the same quality level. I have no doubt NVIDIA sees NGX today as anything but a fixed set of features, but rather an evolving and expanding toolbox of DNNs that can further harness machine learning for the benefit of NVIDIA's traditional visual markets as time goes on.

    AI-enabled DLSS anti-aliasing: There’s great value in any GPU feature that can deliver the same quality in fewer cycles. (Source: NVIDIA)AI-enabled DLSS anti-aliasing: There's great value in any GPU feature that can deliver the same quality in fewer cycles. (Source: NVIDIA)

    And NVIDIA Doubles Down with RT Cores

    Not only did NVIDIA preserve those Tensor Cores when creating Turing, it took the further step of adding multiple instances (one per SM) of an entirely new core design: the RT Core. Specifically, the RT Core takes on a critical ray-tracing computing task, one that when executing on previous GPUs' SMs proved cumbersome, inefficient, and time-intensive. Determining whether a ray (shot from a viewport out into the scene) actually intersects an object (and which triangle on that object's surface) is one of those tasks that a traditional raster-based 3D shader wasn't designed to do, and therefore doesn't do particularly well. With Turing, that job is now left to the RT Cores, freeing up the SMs to spend cycles instead on the 3D shader processing they're more adept at executing.

    Turing’s Streaming Multiprocessor with RT Core. (Source: NVIDIA)Turing's Streaming Multiprocessor with RT Core. (Source: NVIDIA)

    What's the internal micro-architecture of the RT Core look like? Well, NVIDIA hasn't exposed the guts, but given its task — a lot of 3D geometry processing — there's little doubt some high-performance vector and matrix floating-point units form its foundation. What NVIDIA has disclosed is the performance of the RT Core: 10 GigaRays/second. Now, in absolute terms, that's a hard number to assess — like triangles/second in the rasterization world — as it all depends on the workload of each ray measured. The more relevant, apples-to-apples comparison is that rate relative to Pascal's 1.1 GigaRays/second, presumably of the same workload per ray. Given that, Turing is packing a 10X performance improvement processing a crucial and demanding portion of the ray-tracing pipeline.

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    NVIDIA GeForce RTX Explored: What You Need To Know About Turing | killexams.com real questions and Pass4sure dumps

    One of Turing’s main claims to fame is real-time ray tracing. While the GPUs do have capabilities that allow for much faster rendering of ray traced imagery, keep in mind that what we’ve all seen in pre-release games and demos to date are hybrid rendering techniques that leverage traditional rasterization alongside real time ray tracing in some portions of a scene using NVIDIA’s RTX platform software APIs and SDKs. The RT cores and algorithms that accelerate the ray tracing do so using a technique developed by NVIDIA. The GeForce RTX series uses Bounding Volume Hierarchies to efficiently drill down and trace the path of a ray.

    With existing GPUs, using Bounding Volume Hierarchies (or BVH), and emulating what the GeForce RTX series can process, is relatively slow. As we’ve mentioned, the GeForce GTX 1080 Ti is 10x slower than a RTX 2080 Ti with a similar ray tracing workload.

    The GeForce RTX series, however, accelerates the process in hardware. The RT Core includes two specialized units. The first unit does bounding box tests, and the second unit does ray-triangle intersection tests. A ray probe is launched, and if a ray is present in a particular volume, it progresses down the tree and evaluates another, and when there is a hit it’s sent down the pipeline to the SMs for shading. The RT cores in Turing accelerate the BVH traversal and ray-triangle intersection evaluation. While the RT cores are doing their thing, the SMs are also available to perform other rasterization graphics processes or compute work. But what does all of this fancy, highly complex ray-traced graphics processing mean for games? In short, real-time ray tracing allows for dramatically more realistic lighting and reflection rendering, along with how light bounces off and reacts with objects in a scene. Combined with traditional rasterization rendering, ray tracing can provide a much higher level of in-game realism and visual fidelity than previously possible. The screen shot above from Metro Exodus, as well as the live Battlefield V demo above, showcase what real-time ray tracing can do in action and the effects are impressive.Tensor Cores, Machine Learning, NGX, And DLSS DLSS, or Deep Learning Super Sampling, is one of the more interesting and talked about features of NVIDIA’s new Turing architecture and GeForce RTX cards. However, to understand how DLSS works, we need to go back to the hardware and software that powers it, to better understand how NVIDIA gets the job done for impressive visuals with minimal performance impact. First, let’s talk about the Tensor cores in Turing as they relate to DLSS.

    NVIDIA’s new GeForce RTX 2080 Ti, for example, comes equipped with 552 Tensor cores. These are specialized execution units designed for performing tensor generalized matrix operations (math calculations) that are required for Deep Learning. NVIDIA introduced Tensor cores in its Volta GPU architecture but the Tensor cores in Turing have been optimized for inferencing. Inferencing is one of the most fundamental capabilities of machine learning, where the machine analyzes images at the pixel level and literally learns shapes, patterns, coloring, etc., to recognize images very quickly. Is it a dog, a cat, a mouse? You get the idea. And specifically, Turing’s Tensor cores can operate in INT8 and INT4 precision modes for inferencing workloads that don’t the need the FP16 level of precision of Volta Tensor units; or in other words, inferencing for gaming graphics and specifically DLSS (and other new features).

    NVIDIA NGX is the new deep learning-based neural graphics framework of NVIDIA’s RTX platform. NGX uses deep neural networks (DNNs) to perform AI-driven functions that accelerate graphics rendering operations and new features like DLSS (Deep Learning Super Sampling), AI InPainting or content-aware image manipulation, AI Slow-Mo for very high quality slow-motion video, and AI Super Rez smart image resolution resizing. All of these AI-powered features will be accessible via the NGX API which will couple hardware and software together at the driver level.

    NGX services are managed with NVIDIA’s GeForce Experience (GFE or QPX in the case of Quadro cards) software and enabled to run on the GPU, which will not function on NVIDIA architectures prior to Turing. GFE will communicate with NGX to determine what games and apps are installed on the system and will download relevant DNN models for use. NGX DNN models can also interface with CUDA, DirectX and Vulkan to optimize performance while NGX services run on Turing’s Tensor cores. Note, these models are also fairly light-weight and we’re told are only megabytes in size.

    So, that’s the hardware and software behind DLSS, but how does NVIDIA train these Deep Neural Network models in games, for better, faster anti-aliasing? That’s where the rubber will meet the road for DLSS adoption in games. NVIDIA will work with game developers for “free” and have them send them their games to feed the beast, so to speak; the beast being an NVIDIA DGX-1 AI research supercomputer. This powerful AI server will then learn the game visuals frame by frame, comparing them to a “ground truth” golden sample of image quality with 64x super-sampling (64xSS). 64xSS shades at 64 different offsets in a pixel and provides extreme detail, along with ideally anti-aliased rendering. The AI neural network is then tasked with producing an image output, measuring the difference between it and the 64xSS ground truth image quality target, and adjusting its weights accordingly to perfect the image on the next iteration; and the process continues until the model is built.

    The result is both high anti-aliasing image quality in addition to high frame rates. The image above is indicative of what DLSS can achieve in its 2X mode, which is almost indistinguishable from the 64xSS ground truth sample. However, in the video demo depicted in the slide above, you’re seeing DLSS standard mode compared to TAA (Temporal Anti-Aliasing) running at 4K in Epic’s Infiltrator demo using Unreal Engine 4. The machine running TAA at 4K on a GeForce GTX 1080 Ti is slogging it out at just playable frame rates north of 30 FPS. However, the GeForce RTX 2080 Ti with DLSS on (and TAA off) at 4K is able to crest 80 FPS.

    Live and in person, we couldn’t tell the difference in image quality, except that the RTX 2080 Ti powered machine had a much faster frame rate. See for yourself...

    Regardless, the key here is going to be how well NVIDIA is able to execute on getting DLSS modes enabled in new and existing game titles. At first blush, it would seem the feature would be a no-brainer for most devs looking to improve performance and image quality for their games on NVIDIA hardware. In addition, as you can imagine, the more games NVIDIA’s DNNs are fed, the better they’ll get at DLSS, potentially also enabling other image quality features not seen before in games, in the future.Turing Mesh Shaders And Variable Rate Shading NVIDIA’s Turing architecture also introduces new shading techniques that are enabled by a new type of shader technology incorporated into the GPU pipeline. Game developers will now be able to take advantage of Mesh Shaders and Task Shaders in Turing that are much more flexible in terms of how they operate. Both shader types use what NVIDIA calls a “cooperative thread model,” instead of a single-threaded programming model.

    As scene rendering complexity continues to increase, with thousands of individual objects on the screen to apply shader processes to and render, hundreds of thousands of draw calls from the CPU to the GPU can become a bottleneck. However, Turing's new Task Shader stage can now process a list of many objects being sent to the GPU concurrently for processing in parallel, as the Mesh Shaders are then fired to shade triangles to be rasterized.

    You can think of the Task Shader as somewhat of a dispatch pre-processor that alleviates the CPU bottleneck for object draw calls. NVIDIA claims it can enable an increase in the number of objects that can be displayed in real time at gaming frame rates by “an order of magnitude.” In addition, Turing now enables new Variable Rate Shading techniques that can not only allow higher levels of detail, but also boost performance.

    VRS or Variable Rate Shading allows developers much finer-grained control of shading rates in any given scene, when powered by an NVIDIA Turing GPU. Each 16x16 pixel region of a scene can now have different level of shader detail and a different shading rate as a result. The diagram below shows the level of shader detail in 16x16 pixel regions, with the traditional 1x1 blue grid coloring each and every individual pixel. However, with VRS there are 6 additional lower resolution shader rates that can be applied in the 16x16 pixel region, thus also allowing for a possible increase in shader rates in certain areas, because VRS can conserve shader processing resources in other areas of the scene. The image below illustrates different level of detail for the car at full rate (in blue), or near the car once per every four pixels (in green), while the road on the left and right hand sides of the scene can be shaded once every 8 pixels at lower detail (in yellow).

    The car example above is generally what’s referred to as Content Adaptive Shading. NVIDIA claims developers can implement content-based shading rate reductions without modifying their existing rendering pipeline and with only small changes to shader code. However, there are also other adaptive shading techniques like Motion Adaption Shading. Just as it sounds, Motion Adaptive Shading allows for shading rate reduction based on what your eye can actually see for objects in motion. Imagine yourself on a train, looking out the window. Your eyes can fix on an object or a certain area and pick up specific details of it. However, other areas outside that window will be blurred in your periphery, due to the motion of the train. It’s obviously wasteful to shade areas that are motion-blurred at full resolution, so this technique offers a reduced sampling rate, but enough so that it’s visually acceptable to the eye and also at a higher frame rate as a result. Motion Adaptive Shading can also be combined with Content Adaptive Shading for even better shader efficiency.

    Other examples of VRS shading optimization techniques include Foveated Rendering, where shading is done at variable rates based on an area of a scene’s viewing angle to the human eye, and Texture Space shading, where game developers can reuse shader output in areas of a scene that are identical, rather than re-shading those areas wastefully.

    Finally, Turing has better Multiview Rendering at more arbitrary points versus NVIDIA’s previous-generation architectures, with better granularity as well, for VR workloads. As VR devices and head mounted displays gain wider field of views and higher resolution, NVIDIA claims Turing has greater flexibility to take advantage of redundant geometry processing in VR application workloads. Turing’s new Variable Rate Shading techniques, as well as its more flexible Multiview Rendering capabilities will take time for developers to adopt, but the net gain could be over a 20 percent speed-up in graphically rich scenes and game engines, but with comparable or higher image quality as a result of these optimizations.


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